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Effector and regulator: Diverse functions of C. elegans C-type lectin-like domain proteins

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Effector and regulator: Diverse functions of C. elegans C-type lectin-like domain proteins. / Pees, B.; Yang, W.; Kloock, A. et al.
In: PLoS Pathogens, Vol. 17, No. 4, e1009454, 01.04.2021.

Research output: Contribution to Journal/MagazineJournal articlepeer-review

Harvard

Pees, B, Yang, W, Kloock, A, Petersen, C, Peters, L, Fan, L, Friedrichsen, M, Butze, S, Zárate-Potes, A, Schulenburg, H & Dierking, K 2021, 'Effector and regulator: Diverse functions of C. elegans C-type lectin-like domain proteins', PLoS Pathogens, vol. 17, no. 4, e1009454. https://doi.org/10.1371/JOURNAL.PPAT.1009454

APA

Pees, B., Yang, W., Kloock, A., Petersen, C., Peters, L., Fan, L., Friedrichsen, M., Butze, S., Zárate-Potes, A., Schulenburg, H., & Dierking, K. (2021). Effector and regulator: Diverse functions of C. elegans C-type lectin-like domain proteins. PLoS Pathogens, 17(4), Article e1009454. https://doi.org/10.1371/JOURNAL.PPAT.1009454

Vancouver

Pees B, Yang W, Kloock A, Petersen C, Peters L, Fan L et al. Effector and regulator: Diverse functions of C. elegans C-type lectin-like domain proteins. PLoS Pathogens. 2021 Apr 1;17(4):e1009454. doi: 10.1371/JOURNAL.PPAT.1009454

Author

Pees, B. ; Yang, W. ; Kloock, A. et al. / Effector and regulator: Diverse functions of C. elegans C-type lectin-like domain proteins. In: PLoS Pathogens. 2021 ; Vol. 17, No. 4.

Bibtex

@article{b2de481dc89c441697baa046be4ddedf,
title = "Effector and regulator: Diverse functions of C. elegans C-type lectin-like domain proteins",
abstract = "In C. elegans, 283 clec genes encode a highly diverse family of C-type lectin-like domain (CTLD) proteins. Since vertebrate CTLD proteins have characterized functions in defense responses against pathogens and since expression of C. elegans clec genes is pathogen-dependent, it is generally assumed that clec genes function in C. elegans immune defenses. However, little is known about the relative contribution and exact function of CLEC proteins in C. elegans immunity. Here, we focused on the C. elegans clec gene clec-4, whose expression is highly upregulated by pathogen infection, and its paralogs clec-41 and clec-42. We found that, while mutation of clec-4 resulted in enhanced resistance to the Gram-positive pathogen Bacillus thuringiensis MYBt18247 (Bt247), inactivation of clec-41 and clec-42 by RNAi enhanced susceptibility to Bt247. Further analyses revealed that enhanced resistance of clec-4 mutants to Bt247 was due to an increase in feeding cessation on the pathogen and consequently a decrease in pathogen load. Moreover, clec-4 mutants exhibited feeding deficits also on non-pathogenic bacteria that were in part reflected in the clec-4 gene expression profile, which overlapped with gene sets affected by starvation or mutation in nutrient sensing pathways. However, loss of CLEC-4 function only mildly affected life-history traits such as fertility, indicating that clec-4 mutants are not subjected to dietary restriction. While CLEC-4 function appears to be associated with the regulation of feeding behavior, we show that CLEC-41 and CLEC-42 proteins likely function as bona fide immune effector proteins that have bacterial binding and antimicrobial capacities. Together, our results exemplify functional diversification within clec gene paralogs. {\textcopyright} 2021 Pees et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.",
author = "B. Pees and W. Yang and A. Kloock and C. Petersen and L. Peters and L. Fan and M. Friedrichsen and S. Butze and A. Z{\'a}rate-Potes and H. Schulenburg and K. Dierking",
year = "2021",
month = apr,
day = "1",
doi = "10.1371/JOURNAL.PPAT.1009454",
language = "English",
volume = "17",
journal = "PLoS Pathogens",
issn = "1553-7366",
publisher = "Public Library of Science",
number = "4",

}

RIS

TY - JOUR

T1 - Effector and regulator: Diverse functions of C. elegans C-type lectin-like domain proteins

AU - Pees, B.

AU - Yang, W.

AU - Kloock, A.

AU - Petersen, C.

AU - Peters, L.

AU - Fan, L.

AU - Friedrichsen, M.

AU - Butze, S.

AU - Zárate-Potes, A.

AU - Schulenburg, H.

AU - Dierking, K.

PY - 2021/4/1

Y1 - 2021/4/1

N2 - In C. elegans, 283 clec genes encode a highly diverse family of C-type lectin-like domain (CTLD) proteins. Since vertebrate CTLD proteins have characterized functions in defense responses against pathogens and since expression of C. elegans clec genes is pathogen-dependent, it is generally assumed that clec genes function in C. elegans immune defenses. However, little is known about the relative contribution and exact function of CLEC proteins in C. elegans immunity. Here, we focused on the C. elegans clec gene clec-4, whose expression is highly upregulated by pathogen infection, and its paralogs clec-41 and clec-42. We found that, while mutation of clec-4 resulted in enhanced resistance to the Gram-positive pathogen Bacillus thuringiensis MYBt18247 (Bt247), inactivation of clec-41 and clec-42 by RNAi enhanced susceptibility to Bt247. Further analyses revealed that enhanced resistance of clec-4 mutants to Bt247 was due to an increase in feeding cessation on the pathogen and consequently a decrease in pathogen load. Moreover, clec-4 mutants exhibited feeding deficits also on non-pathogenic bacteria that were in part reflected in the clec-4 gene expression profile, which overlapped with gene sets affected by starvation or mutation in nutrient sensing pathways. However, loss of CLEC-4 function only mildly affected life-history traits such as fertility, indicating that clec-4 mutants are not subjected to dietary restriction. While CLEC-4 function appears to be associated with the regulation of feeding behavior, we show that CLEC-41 and CLEC-42 proteins likely function as bona fide immune effector proteins that have bacterial binding and antimicrobial capacities. Together, our results exemplify functional diversification within clec gene paralogs. © 2021 Pees et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

AB - In C. elegans, 283 clec genes encode a highly diverse family of C-type lectin-like domain (CTLD) proteins. Since vertebrate CTLD proteins have characterized functions in defense responses against pathogens and since expression of C. elegans clec genes is pathogen-dependent, it is generally assumed that clec genes function in C. elegans immune defenses. However, little is known about the relative contribution and exact function of CLEC proteins in C. elegans immunity. Here, we focused on the C. elegans clec gene clec-4, whose expression is highly upregulated by pathogen infection, and its paralogs clec-41 and clec-42. We found that, while mutation of clec-4 resulted in enhanced resistance to the Gram-positive pathogen Bacillus thuringiensis MYBt18247 (Bt247), inactivation of clec-41 and clec-42 by RNAi enhanced susceptibility to Bt247. Further analyses revealed that enhanced resistance of clec-4 mutants to Bt247 was due to an increase in feeding cessation on the pathogen and consequently a decrease in pathogen load. Moreover, clec-4 mutants exhibited feeding deficits also on non-pathogenic bacteria that were in part reflected in the clec-4 gene expression profile, which overlapped with gene sets affected by starvation or mutation in nutrient sensing pathways. However, loss of CLEC-4 function only mildly affected life-history traits such as fertility, indicating that clec-4 mutants are not subjected to dietary restriction. While CLEC-4 function appears to be associated with the regulation of feeding behavior, we show that CLEC-41 and CLEC-42 proteins likely function as bona fide immune effector proteins that have bacterial binding and antimicrobial capacities. Together, our results exemplify functional diversification within clec gene paralogs. © 2021 Pees et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

U2 - 10.1371/JOURNAL.PPAT.1009454

DO - 10.1371/JOURNAL.PPAT.1009454

M3 - Journal article

VL - 17

JO - PLoS Pathogens

JF - PLoS Pathogens

SN - 1553-7366

IS - 4

M1 - e1009454

ER -