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Androgen receptors are acquired by healthy postmenopausal endometrial epithelium and their subsequent loss in endometrial cancer is associated with poor survival

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Androgen receptors are acquired by healthy postmenopausal endometrial epithelium and their subsequent loss in endometrial cancer is associated with poor survival. / Kamal, A. M.; Bulmer, J. N.; DeCruze, S. B. et al.
In: British Journal of Cancer, Vol. 114, 15.03.2016, p. 688-696.

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Harvard

Kamal, AM, Bulmer, JN, DeCruze, SB, Stringfellow, HF, Martin-Hirsch, PL & Hapangama, D 2016, 'Androgen receptors are acquired by healthy postmenopausal endometrial epithelium and their subsequent loss in endometrial cancer is associated with poor survival', British Journal of Cancer, vol. 114, pp. 688-696. https://doi.org/10.1038/bjc.2016.16

APA

Kamal, A. M., Bulmer, J. N., DeCruze, S. B., Stringfellow, H. F., Martin-Hirsch, P. L., & Hapangama, D. (2016). Androgen receptors are acquired by healthy postmenopausal endometrial epithelium and their subsequent loss in endometrial cancer is associated with poor survival. British Journal of Cancer, 114, 688-696. https://doi.org/10.1038/bjc.2016.16

Vancouver

Kamal AM, Bulmer JN, DeCruze SB, Stringfellow HF, Martin-Hirsch PL, Hapangama D. Androgen receptors are acquired by healthy postmenopausal endometrial epithelium and their subsequent loss in endometrial cancer is associated with poor survival. British Journal of Cancer. 2016 Mar 15;114:688-696. Epub 2016 Mar 1. doi: 10.1038/bjc.2016.16

Author

Kamal, A. M. ; Bulmer, J. N. ; DeCruze, S. B. et al. / Androgen receptors are acquired by healthy postmenopausal endometrial epithelium and their subsequent loss in endometrial cancer is associated with poor survival. In: British Journal of Cancer. 2016 ; Vol. 114. pp. 688-696.

Bibtex

@article{2bec1ae091624e4d94f850013e646c32,
title = "Androgen receptors are acquired by healthy postmenopausal endometrial epithelium and their subsequent loss in endometrial cancer is associated with poor survival",
abstract = "background: Endometrial cancer (EC) is a hormone-driven disease, and androgen receptor (AR) expression in high-grade EC (HGEC) and metastatic EC has not yet been described. methods: The expression pattern and prognostic value of AR in relation to oestrogen (ERα and ERβ) and progesterone (PR) receptors, and the proliferation marker Ki67 in all EC subtypes (n=85) were compared with that of healthy and hyperplastic endometrium, using immunohistochemisty and qPCR. results: Compared with proliferative endometrium, postmenopausal endometrtial epithelium showed significantly higher expression of AR (P<0.001) and ERα (P=0.035), which persisted in hyperplastic epithelium and in low-grade EC (LGEC). High-grade EC showed a significant loss of AR (P<0.0001), PR (P<0.0001) and ERβ (P<0.035) compared with LGEC, whilst maintaining weak to moderate ERα. Unlike PR, AR expression in metastatic lesions was significantly (P=0.039) higher than that in primary tumours. Androgen receptor expression correlated with favourable clinicopathological features and a lower proliferation index. Loss of AR, with/without the loss of PR was associated with a significantly lower disease-free survival (P<0.0001, P<0.0001, respectively). conclusions: Postmenopausal endometrial epithelium acquires AR whilst preserving other steroid hormone receptors. Loss of AR, PR with retention of ERα and ERβ may promote the unrestrained growth of HGEC. Androgen receptor may therefore be a clinically relevant prognostic indicator and a potential therapeutic target in EC.",
keywords = "androgen receptor (AR), endometrial cancer, postmenopausal endometrium, metastatic lesions, ERβ, ERα, PR, outcome",
author = "Kamal, {A. M.} and Bulmer, {J. N.} and DeCruze, {S. B.} and Stringfellow, {Helen F.} and Martin-Hirsch, {Pierre Leonard} and Dharani Hapangama",
year = "2016",
month = mar,
day = "15",
doi = "10.1038/bjc.2016.16",
language = "English",
volume = "114",
pages = "688--696",
journal = "British Journal of Cancer",
issn = "0007-0920",
publisher = "Nature Publishing Group",

}

RIS

TY - JOUR

T1 - Androgen receptors are acquired by healthy postmenopausal endometrial epithelium and their subsequent loss in endometrial cancer is associated with poor survival

AU - Kamal, A. M.

AU - Bulmer, J. N.

AU - DeCruze, S. B.

AU - Stringfellow, Helen F.

AU - Martin-Hirsch, Pierre Leonard

AU - Hapangama, Dharani

PY - 2016/3/15

Y1 - 2016/3/15

N2 - background: Endometrial cancer (EC) is a hormone-driven disease, and androgen receptor (AR) expression in high-grade EC (HGEC) and metastatic EC has not yet been described. methods: The expression pattern and prognostic value of AR in relation to oestrogen (ERα and ERβ) and progesterone (PR) receptors, and the proliferation marker Ki67 in all EC subtypes (n=85) were compared with that of healthy and hyperplastic endometrium, using immunohistochemisty and qPCR. results: Compared with proliferative endometrium, postmenopausal endometrtial epithelium showed significantly higher expression of AR (P<0.001) and ERα (P=0.035), which persisted in hyperplastic epithelium and in low-grade EC (LGEC). High-grade EC showed a significant loss of AR (P<0.0001), PR (P<0.0001) and ERβ (P<0.035) compared with LGEC, whilst maintaining weak to moderate ERα. Unlike PR, AR expression in metastatic lesions was significantly (P=0.039) higher than that in primary tumours. Androgen receptor expression correlated with favourable clinicopathological features and a lower proliferation index. Loss of AR, with/without the loss of PR was associated with a significantly lower disease-free survival (P<0.0001, P<0.0001, respectively). conclusions: Postmenopausal endometrial epithelium acquires AR whilst preserving other steroid hormone receptors. Loss of AR, PR with retention of ERα and ERβ may promote the unrestrained growth of HGEC. Androgen receptor may therefore be a clinically relevant prognostic indicator and a potential therapeutic target in EC.

AB - background: Endometrial cancer (EC) is a hormone-driven disease, and androgen receptor (AR) expression in high-grade EC (HGEC) and metastatic EC has not yet been described. methods: The expression pattern and prognostic value of AR in relation to oestrogen (ERα and ERβ) and progesterone (PR) receptors, and the proliferation marker Ki67 in all EC subtypes (n=85) were compared with that of healthy and hyperplastic endometrium, using immunohistochemisty and qPCR. results: Compared with proliferative endometrium, postmenopausal endometrtial epithelium showed significantly higher expression of AR (P<0.001) and ERα (P=0.035), which persisted in hyperplastic epithelium and in low-grade EC (LGEC). High-grade EC showed a significant loss of AR (P<0.0001), PR (P<0.0001) and ERβ (P<0.035) compared with LGEC, whilst maintaining weak to moderate ERα. Unlike PR, AR expression in metastatic lesions was significantly (P=0.039) higher than that in primary tumours. Androgen receptor expression correlated with favourable clinicopathological features and a lower proliferation index. Loss of AR, with/without the loss of PR was associated with a significantly lower disease-free survival (P<0.0001, P<0.0001, respectively). conclusions: Postmenopausal endometrial epithelium acquires AR whilst preserving other steroid hormone receptors. Loss of AR, PR with retention of ERα and ERβ may promote the unrestrained growth of HGEC. Androgen receptor may therefore be a clinically relevant prognostic indicator and a potential therapeutic target in EC.

KW - androgen receptor (AR)

KW - endometrial cancer

KW - postmenopausal endometrium

KW - metastatic lesions

KW - ERβ

KW - ERα

KW - PR

KW - outcome

U2 - 10.1038/bjc.2016.16

DO - 10.1038/bjc.2016.16

M3 - Journal article

VL - 114

SP - 688

EP - 696

JO - British Journal of Cancer

JF - British Journal of Cancer

SN - 0007-0920

ER -