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Glucose transport regulation by p210 Bcr-Abl in a chronic myeloid leukaemia model.

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Glucose transport regulation by p210 Bcr-Abl in a chronic myeloid leukaemia model. / Bentley, J.; Walker, I.; McIntosh, E. et al.
In: British Journal of Haematology, Vol. 112, No. 1, 01.2001, p. 212-215.

Research output: Contribution to Journal/Magazine › Journal article › peer-review

Harvard

Bentley, J, Walker, I, McIntosh, E, Whetton, AD, Owen-Lynch, PJ & Baldwin, SA 2001, 'Glucose transport regulation by p210 Bcr-Abl in a chronic myeloid leukaemia model.', British Journal of Haematology, vol. 112, no. 1, pp. 212-215. https://doi.org/10.1046/j.1365-2141.2001.02428.x

APA

Bentley, J., Walker, I., McIntosh, E., Whetton, A. D., Owen-Lynch, P. J., & Baldwin, S. A. (2001). Glucose transport regulation by p210 Bcr-Abl in a chronic myeloid leukaemia model. British Journal of Haematology, 112(1), 212-215. https://doi.org/10.1046/j.1365-2141.2001.02428.x

Vancouver

Bentley J, Walker I, McIntosh E, Whetton AD, Owen-Lynch PJ, Baldwin SA. Glucose transport regulation by p210 Bcr-Abl in a chronic myeloid leukaemia model. British Journal of Haematology. 2001 Jan;112(1):212-215. doi: 10.1046/j.1365-2141.2001.02428.x

Author

Bentley, J. ; Walker, I. ; McIntosh, E. et al. / Glucose transport regulation by p210 Bcr-Abl in a chronic myeloid leukaemia model. In: British Journal of Haematology. 2001 ; Vol. 112, No. 1. pp. 212-215.

Bibtex

@article{41697ac5b419408c884c31d95be0f18e,
title = "Glucose transport regulation by p210 Bcr-Abl in a chronic myeloid leukaemia model.",
abstract = "The regulation of nutrient transport by both cytokines and oncogenes has been linked to haemopoietic cell survival. In this study, we found that activation of Bcr–Abl protein tyrosine kinase was associated with the stimulation of glucose transport in the multipotent haemopoietic cell line FDCP-mix, a cell model for chronic-phase chronic myeloid leukaemia (CML). Bcr–Abl upregulation of glucose transport was mediated by phosphatidylinositol-3-kinase. The observation that Bcr–Abl can regulate glucose transport in a CML cell model raises the possibility that glucose transport regulation may have a role to play in the aberrant survival of stem cells in the chronic phase of CML.",
author = "J. Bentley and I. Walker and E. McIntosh and Whetton, {Anthony D.} and Owen-Lynch, {P. Jane} and Baldwin, {S. A.}",
year = "2001",
month = jan,
doi = "10.1046/j.1365-2141.2001.02428.x",
language = "English",
volume = "112",
pages = "212--215",
journal = "British Journal of Haematology",
issn = "0007-1048",
publisher = "Wiley-Blackwell",
number = "1",

}

RIS

TY - JOUR

T1 - Glucose transport regulation by p210 Bcr-Abl in a chronic myeloid leukaemia model.

AU - Bentley, J.

AU - Walker, I.

AU - McIntosh, E.

AU - Whetton, Anthony D.

AU - Owen-Lynch, P. Jane

AU - Baldwin, S. A.

PY - 2001/1

Y1 - 2001/1

N2 - The regulation of nutrient transport by both cytokines and oncogenes has been linked to haemopoietic cell survival. In this study, we found that activation of Bcr–Abl protein tyrosine kinase was associated with the stimulation of glucose transport in the multipotent haemopoietic cell line FDCP-mix, a cell model for chronic-phase chronic myeloid leukaemia (CML). Bcr–Abl upregulation of glucose transport was mediated by phosphatidylinositol-3-kinase. The observation that Bcr–Abl can regulate glucose transport in a CML cell model raises the possibility that glucose transport regulation may have a role to play in the aberrant survival of stem cells in the chronic phase of CML.

AB - The regulation of nutrient transport by both cytokines and oncogenes has been linked to haemopoietic cell survival. In this study, we found that activation of Bcr–Abl protein tyrosine kinase was associated with the stimulation of glucose transport in the multipotent haemopoietic cell line FDCP-mix, a cell model for chronic-phase chronic myeloid leukaemia (CML). Bcr–Abl upregulation of glucose transport was mediated by phosphatidylinositol-3-kinase. The observation that Bcr–Abl can regulate glucose transport in a CML cell model raises the possibility that glucose transport regulation may have a role to play in the aberrant survival of stem cells in the chronic phase of CML.

U2 - 10.1046/j.1365-2141.2001.02428.x

DO - 10.1046/j.1365-2141.2001.02428.x

M3 - Journal article

VL - 112

SP - 212

EP - 215

JO - British Journal of Haematology

JF - British Journal of Haematology

SN - 0007-1048

IS - 1

ER -