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Identification of 13 novel NLRP7 mutations in 20 families with recurrent hydatidiform mole; missense mutations cluster in the leucine-rich region

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Identification of 13 novel NLRP7 mutations in 20 families with recurrent hydatidiform mole; missense mutations cluster in the leucine-rich region. / Wang, C. M.; Dixon, P. H.; Decordova, S. et al.
In: Journal of Medical Genetics, Vol. 46, No. 8, 2009, p. 569-575.

Research output: Contribution to Journal/MagazineJournal articlepeer-review

Harvard

Wang, CM, Dixon, PH, Decordova, S, Hodges, M, Seibre, NJ, Ozalp, S, Fallahian, M, Sensi, A, Ashrafi, F, Repiska, V, Zhao, J, Xiang, Y, Savage, PM, Seckl, MJ & Fisher, RA 2009, 'Identification of 13 novel NLRP7 mutations in 20 families with recurrent hydatidiform mole; missense mutations cluster in the leucine-rich region', Journal of Medical Genetics, vol. 46, no. 8, pp. 569-575. https://doi.org/10.1136/jmg.2008.064196

APA

Wang, C. M., Dixon, P. H., Decordova, S., Hodges, M., Seibre, N. J., Ozalp, S., Fallahian, M., Sensi, A., Ashrafi, F., Repiska, V., Zhao, J., Xiang, Y., Savage, P. M., Seckl, M. J., & Fisher, R. A. (2009). Identification of 13 novel NLRP7 mutations in 20 families with recurrent hydatidiform mole; missense mutations cluster in the leucine-rich region. Journal of Medical Genetics, 46(8), 569-575. https://doi.org/10.1136/jmg.2008.064196

Vancouver

Wang CM, Dixon PH, Decordova S, Hodges M, Seibre NJ, Ozalp S et al. Identification of 13 novel NLRP7 mutations in 20 families with recurrent hydatidiform mole; missense mutations cluster in the leucine-rich region. Journal of Medical Genetics. 2009;46(8):569-575. Epub 2009 Feb 25. doi: 10.1136/jmg.2008.064196

Author

Wang, C. M. ; Dixon, P. H. ; Decordova, S. et al. / Identification of 13 novel NLRP7 mutations in 20 families with recurrent hydatidiform mole; missense mutations cluster in the leucine-rich region. In: Journal of Medical Genetics. 2009 ; Vol. 46, No. 8. pp. 569-575.

Bibtex

@article{b8dfda019bf04578b0d60539c4e2b2d1,
title = "Identification of 13 novel NLRP7 mutations in 20 families with recurrent hydatidiform mole; missense mutations cluster in the leucine-rich region",
abstract = "Background: NLRP7 (NALP7) has recently been identified as the causative gene for familial recurrent hydatidiform mole (FRHM), a rare autosomal recessive condition in which affected women have recurrent molar pregnancies of diploid biparental origin. To date only a small number of affected families have been described. Our objectives were to investigate the diversity of mutations and their localisation to one or both isoforms of NLRP7, by screening a large series of women with FRHM and to examine the normal expression of NLRP7 in ovarian tissue.Methods: Fluorescent microsatellite genotyping of molar tissue was used to establish a diagnosis of FRHM. Twenty families were subsequently screened for mutations in NLRP7 using DNA sequencing. Expression of NLRP7 in the ovary was examined by immunohistochemical staining.Results: 16 different mutations were identified in the study, 13 of which were novel. Missense mutations were found to be present in transcript variant 2 of NLRP7 and cluster in the leucine-rich region (LRR). A man with two affected sisters and homozygous for the p.R693P mutation had normal reproductive outcomes. In the normal human ovary, NLRP7 expression is confined to the oocytes and present at all stages from primordial to tertiary follicles.Conclusion: 13 novel mutations in NLRP7 were identified. We confirm that mutations in NLRP7 affect female but not male reproduction, and provide evidence that transcript variant 2 of NLRP7 is the important isoform in this condition. The mutation clustering seen confirms that the LRR is critical for normal functioning of NLRP7.",
keywords = "Mutation, NRLP7, recurant hydatidiform mole",
author = "Wang, {C. M.} and Dixon, {P. H.} and S. Decordova and Matt Hodges and Seibre, {N. J.} and S. Ozalp and M. Fallahian and A. Sensi and F. Ashrafi and V. Repiska and J. Zhao and Y. Xiang and Savage, {P. M.} and Seckl, {M. J.} and Fisher, {R. A.}",
year = "2009",
doi = "10.1136/jmg.2008.064196",
language = "English",
volume = "46",
pages = "569--575",
journal = "Journal of Medical Genetics",
issn = "1468-6244",
publisher = "BMJ Publishing Group",
number = "8",

}

RIS

TY - JOUR

T1 - Identification of 13 novel NLRP7 mutations in 20 families with recurrent hydatidiform mole; missense mutations cluster in the leucine-rich region

AU - Wang, C. M.

AU - Dixon, P. H.

AU - Decordova, S.

AU - Hodges, Matt

AU - Seibre, N. J.

AU - Ozalp, S.

AU - Fallahian, M.

AU - Sensi, A.

AU - Ashrafi, F.

AU - Repiska, V.

AU - Zhao, J.

AU - Xiang, Y.

AU - Savage, P. M.

AU - Seckl, M. J.

AU - Fisher, R. A.

PY - 2009

Y1 - 2009

N2 - Background: NLRP7 (NALP7) has recently been identified as the causative gene for familial recurrent hydatidiform mole (FRHM), a rare autosomal recessive condition in which affected women have recurrent molar pregnancies of diploid biparental origin. To date only a small number of affected families have been described. Our objectives were to investigate the diversity of mutations and their localisation to one or both isoforms of NLRP7, by screening a large series of women with FRHM and to examine the normal expression of NLRP7 in ovarian tissue.Methods: Fluorescent microsatellite genotyping of molar tissue was used to establish a diagnosis of FRHM. Twenty families were subsequently screened for mutations in NLRP7 using DNA sequencing. Expression of NLRP7 in the ovary was examined by immunohistochemical staining.Results: 16 different mutations were identified in the study, 13 of which were novel. Missense mutations were found to be present in transcript variant 2 of NLRP7 and cluster in the leucine-rich region (LRR). A man with two affected sisters and homozygous for the p.R693P mutation had normal reproductive outcomes. In the normal human ovary, NLRP7 expression is confined to the oocytes and present at all stages from primordial to tertiary follicles.Conclusion: 13 novel mutations in NLRP7 were identified. We confirm that mutations in NLRP7 affect female but not male reproduction, and provide evidence that transcript variant 2 of NLRP7 is the important isoform in this condition. The mutation clustering seen confirms that the LRR is critical for normal functioning of NLRP7.

AB - Background: NLRP7 (NALP7) has recently been identified as the causative gene for familial recurrent hydatidiform mole (FRHM), a rare autosomal recessive condition in which affected women have recurrent molar pregnancies of diploid biparental origin. To date only a small number of affected families have been described. Our objectives were to investigate the diversity of mutations and their localisation to one or both isoforms of NLRP7, by screening a large series of women with FRHM and to examine the normal expression of NLRP7 in ovarian tissue.Methods: Fluorescent microsatellite genotyping of molar tissue was used to establish a diagnosis of FRHM. Twenty families were subsequently screened for mutations in NLRP7 using DNA sequencing. Expression of NLRP7 in the ovary was examined by immunohistochemical staining.Results: 16 different mutations were identified in the study, 13 of which were novel. Missense mutations were found to be present in transcript variant 2 of NLRP7 and cluster in the leucine-rich region (LRR). A man with two affected sisters and homozygous for the p.R693P mutation had normal reproductive outcomes. In the normal human ovary, NLRP7 expression is confined to the oocytes and present at all stages from primordial to tertiary follicles.Conclusion: 13 novel mutations in NLRP7 were identified. We confirm that mutations in NLRP7 affect female but not male reproduction, and provide evidence that transcript variant 2 of NLRP7 is the important isoform in this condition. The mutation clustering seen confirms that the LRR is critical for normal functioning of NLRP7.

KW - Mutation

KW - NRLP7

KW - recurant hydatidiform mole

U2 - 10.1136/jmg.2008.064196

DO - 10.1136/jmg.2008.064196

M3 - Journal article

VL - 46

SP - 569

EP - 575

JO - Journal of Medical Genetics

JF - Journal of Medical Genetics

SN - 1468-6244

IS - 8

ER -