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Mistargeting of aggregation prone mitochondrial proteins activates a nucleus-mediated posttranscriptional quality control pathway in trypanosomes

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Mistargeting of aggregation prone mitochondrial proteins activates a nucleus-mediated posttranscriptional quality control pathway in trypanosomes. / Dewar, Caroline E.; Oeljeklaus, Silke; Mani, Jan et al.
In: Nature Communications, Vol. 13, 3084, 02.06.2022.

Research output: Contribution to Journal/MagazineJournal articlepeer-review

Harvard

Dewar, CE, Oeljeklaus, S, Mani, J, Muehlhaeuser, WWD, von Kaenel, C, Zimmermann, J, Ochsenreiter, T, Warscheid, B & Schneider, A 2022, 'Mistargeting of aggregation prone mitochondrial proteins activates a nucleus-mediated posttranscriptional quality control pathway in trypanosomes', Nature Communications, vol. 13, 3084. https://doi.org/10.1038/S41467-022-30748-Z

APA

Dewar, C. E., Oeljeklaus, S., Mani, J., Muehlhaeuser, W. W. D., von Kaenel, C., Zimmermann, J., Ochsenreiter, T., Warscheid, B., & Schneider, A. (2022). Mistargeting of aggregation prone mitochondrial proteins activates a nucleus-mediated posttranscriptional quality control pathway in trypanosomes. Nature Communications, 13, Article 3084. https://doi.org/10.1038/S41467-022-30748-Z

Vancouver

Dewar CE, Oeljeklaus S, Mani J, Muehlhaeuser WWD, von Kaenel C, Zimmermann J et al. Mistargeting of aggregation prone mitochondrial proteins activates a nucleus-mediated posttranscriptional quality control pathway in trypanosomes. Nature Communications. 2022 Jun 2;13:3084. doi: 10.1038/S41467-022-30748-Z

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Bibtex

@article{fd320899df104b669a72f9bf386f11b7,
title = "Mistargeting of aggregation prone mitochondrial proteins activates a nucleus-mediated posttranscriptional quality control pathway in trypanosomes",
abstract = "Mitochondrial protein import in the parasitic protozoan Trypanosoma brucei is mediated by the atypical outer membrane translocase, ATOM. It consists of seven subunits including ATOM69, the import receptor for hydrophobic proteins. Ablation of ATOM69, but not of any other subunit, triggers a unique quality control pathway resulting in the proteasomal degradation of non-imported mitochondrial proteins. The process requires a protein of unknown function, an E3 ubiquitin ligase and the ubiquitin-like protein (TbUbL1), which all are recruited to the mitochondrion upon ATOM69 depletion. TbUbL1 is a nuclear protein, a fraction of which is released to the cytosol upon triggering of the pathway. Nuclear release is essential as cytosolic TbUbL1 can bind mislocalised mitochondrial proteins and likely transfers them to the proteasome. Mitochondrial quality control has previously been studied in yeast and metazoans. Finding such a pathway in the highly diverged trypanosomes suggests such pathways are an obligate feature of all eukaryotes.",
author = "Dewar, {Caroline E.} and Silke Oeljeklaus and Jan Mani and Muehlhaeuser, {Wignand W. D.} and {von Kaenel}, Corinne and Johannes Zimmermann and Torsten Ochsenreiter and Bettina Warscheid and Andre Schneider",
year = "2022",
month = jun,
day = "2",
doi = "10.1038/S41467-022-30748-Z",
language = "English",
volume = "13",
journal = "Nature Communications",
issn = "2041-1723",
publisher = "Nature Publishing Group",

}

RIS

TY - JOUR

T1 - Mistargeting of aggregation prone mitochondrial proteins activates a nucleus-mediated posttranscriptional quality control pathway in trypanosomes

AU - Dewar, Caroline E.

AU - Oeljeklaus, Silke

AU - Mani, Jan

AU - Muehlhaeuser, Wignand W. D.

AU - von Kaenel, Corinne

AU - Zimmermann, Johannes

AU - Ochsenreiter, Torsten

AU - Warscheid, Bettina

AU - Schneider, Andre

PY - 2022/6/2

Y1 - 2022/6/2

N2 - Mitochondrial protein import in the parasitic protozoan Trypanosoma brucei is mediated by the atypical outer membrane translocase, ATOM. It consists of seven subunits including ATOM69, the import receptor for hydrophobic proteins. Ablation of ATOM69, but not of any other subunit, triggers a unique quality control pathway resulting in the proteasomal degradation of non-imported mitochondrial proteins. The process requires a protein of unknown function, an E3 ubiquitin ligase and the ubiquitin-like protein (TbUbL1), which all are recruited to the mitochondrion upon ATOM69 depletion. TbUbL1 is a nuclear protein, a fraction of which is released to the cytosol upon triggering of the pathway. Nuclear release is essential as cytosolic TbUbL1 can bind mislocalised mitochondrial proteins and likely transfers them to the proteasome. Mitochondrial quality control has previously been studied in yeast and metazoans. Finding such a pathway in the highly diverged trypanosomes suggests such pathways are an obligate feature of all eukaryotes.

AB - Mitochondrial protein import in the parasitic protozoan Trypanosoma brucei is mediated by the atypical outer membrane translocase, ATOM. It consists of seven subunits including ATOM69, the import receptor for hydrophobic proteins. Ablation of ATOM69, but not of any other subunit, triggers a unique quality control pathway resulting in the proteasomal degradation of non-imported mitochondrial proteins. The process requires a protein of unknown function, an E3 ubiquitin ligase and the ubiquitin-like protein (TbUbL1), which all are recruited to the mitochondrion upon ATOM69 depletion. TbUbL1 is a nuclear protein, a fraction of which is released to the cytosol upon triggering of the pathway. Nuclear release is essential as cytosolic TbUbL1 can bind mislocalised mitochondrial proteins and likely transfers them to the proteasome. Mitochondrial quality control has previously been studied in yeast and metazoans. Finding such a pathway in the highly diverged trypanosomes suggests such pathways are an obligate feature of all eukaryotes.

UR - https://publons.com/wos-op/publon/62612555/

U2 - 10.1038/S41467-022-30748-Z

DO - 10.1038/S41467-022-30748-Z

M3 - Journal article

VL - 13

JO - Nature Communications

JF - Nature Communications

SN - 2041-1723

M1 - 3084

ER -