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The effect of general anaesthetics on brain lactate release.

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The effect of general anaesthetics on brain lactate release. / Hadjihambi, Anna; Karagiannis, A; Theparambil, SM et al.
In: European Journal of Pharmacology, Vol. 881, 173188, 15.08.2020.

Research output: Contribution to Journal/MagazineJournal articlepeer-review

Harvard

Hadjihambi, A, Karagiannis, A, Theparambil, SM, Gareth, LAPFRCAFFICMFHEA & Gourine, AV 2020, 'The effect of general anaesthetics on brain lactate release.', European Journal of Pharmacology, vol. 881, 173188. https://doi.org/10.1016/j.ejphar.2020.173188

APA

Hadjihambi, A., Karagiannis, A., Theparambil, SM., Gareth, L. A. P. FRCA. FFICM. FHEA., & Gourine, AV. (2020). The effect of general anaesthetics on brain lactate release. European Journal of Pharmacology, 881, Article 173188. https://doi.org/10.1016/j.ejphar.2020.173188

Vancouver

Hadjihambi A, Karagiannis A, Theparambil SM, Gareth LAPFRCAFFICMFHEA, Gourine AV. The effect of general anaesthetics on brain lactate release. European Journal of Pharmacology. 2020 Aug 15;881:173188. doi: 10.1016/j.ejphar.2020.173188

Author

Hadjihambi, Anna ; Karagiannis, A ; Theparambil, SM et al. / The effect of general anaesthetics on brain lactate release. In: European Journal of Pharmacology. 2020 ; Vol. 881.

Bibtex

@article{6e7d601c134243ebbfb33a7abb821664,
title = "The effect of general anaesthetics on brain lactate release.",
abstract = "The effects of anaesthetic agents on brain energy metabolism may explain their shared neurophysiological actions but remain poorly understood. The brain lactate shuttle hypothesis proposes that lactate, provided by astrocytes, is an important neuronal energy substrate. Here we tested the hypothesis that anaesthetic agents impair the brain lactate shuttle by interfering with astrocytic glycolysis. Lactate biosensors were used to record changes in lactate release by adult rat brainstem and cortical slices in response to thiopental, propofol and etomidate. Changes in cytosolic nicotinamide adenine dinucleotide reduced (NADH) and oxidized (NAD+) ratio as a measure of glycolytic rate were recorded in cultured astrocytes. It was found that in brainstem slices thiopental, propofol and etomidate reduced lactate release by 7.4 ± 3.6% (P +] ratio in astrocytes, indicative of a reduction in glycolytic rate. These data suggest that anaesthetic agents inhibit astrocytic glycolysis and reduce the level of extracellular lactate in the brain. Similar reductions in brain lactate release occur during natural state of sleep, suggesting that general anaesthesia may recapitulate some of the effects of sleep on brain energy metabolism.",
author = "Anna Hadjihambi and A Karagiannis and SM Theparambil and Gareth, {L. Ackland PhD FRCA FFICM FHEA} and AV Gourine",
year = "2020",
month = aug,
day = "15",
doi = "10.1016/j.ejphar.2020.173188",
language = "English",
volume = "881",
journal = "European Journal of Pharmacology",
issn = "0014-2999",
publisher = "Elsevier",

}

RIS

TY - JOUR

T1 - The effect of general anaesthetics on brain lactate release.

AU - Hadjihambi, Anna

AU - Karagiannis, A

AU - Theparambil, SM

AU - Gareth, L. Ackland PhD FRCA FFICM FHEA

AU - Gourine, AV

PY - 2020/8/15

Y1 - 2020/8/15

N2 - The effects of anaesthetic agents on brain energy metabolism may explain their shared neurophysiological actions but remain poorly understood. The brain lactate shuttle hypothesis proposes that lactate, provided by astrocytes, is an important neuronal energy substrate. Here we tested the hypothesis that anaesthetic agents impair the brain lactate shuttle by interfering with astrocytic glycolysis. Lactate biosensors were used to record changes in lactate release by adult rat brainstem and cortical slices in response to thiopental, propofol and etomidate. Changes in cytosolic nicotinamide adenine dinucleotide reduced (NADH) and oxidized (NAD+) ratio as a measure of glycolytic rate were recorded in cultured astrocytes. It was found that in brainstem slices thiopental, propofol and etomidate reduced lactate release by 7.4 ± 3.6% (P +] ratio in astrocytes, indicative of a reduction in glycolytic rate. These data suggest that anaesthetic agents inhibit astrocytic glycolysis and reduce the level of extracellular lactate in the brain. Similar reductions in brain lactate release occur during natural state of sleep, suggesting that general anaesthesia may recapitulate some of the effects of sleep on brain energy metabolism.

AB - The effects of anaesthetic agents on brain energy metabolism may explain their shared neurophysiological actions but remain poorly understood. The brain lactate shuttle hypothesis proposes that lactate, provided by astrocytes, is an important neuronal energy substrate. Here we tested the hypothesis that anaesthetic agents impair the brain lactate shuttle by interfering with astrocytic glycolysis. Lactate biosensors were used to record changes in lactate release by adult rat brainstem and cortical slices in response to thiopental, propofol and etomidate. Changes in cytosolic nicotinamide adenine dinucleotide reduced (NADH) and oxidized (NAD+) ratio as a measure of glycolytic rate were recorded in cultured astrocytes. It was found that in brainstem slices thiopental, propofol and etomidate reduced lactate release by 7.4 ± 3.6% (P +] ratio in astrocytes, indicative of a reduction in glycolytic rate. These data suggest that anaesthetic agents inhibit astrocytic glycolysis and reduce the level of extracellular lactate in the brain. Similar reductions in brain lactate release occur during natural state of sleep, suggesting that general anaesthesia may recapitulate some of the effects of sleep on brain energy metabolism.

U2 - 10.1016/j.ejphar.2020.173188

DO - 10.1016/j.ejphar.2020.173188

M3 - Journal article

C2 - 32439258

VL - 881

JO - European Journal of Pharmacology

JF - European Journal of Pharmacology

SN - 0014-2999

M1 - 173188

ER -