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Thermal biology of mosquito‐borne disease

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Thermal biology of mosquito‐borne disease. / Mordecai, Erin A.; Caldwell, Jamie M.; Grossman, Marissa K. et al.
In: Ecology Letters, Vol. 22, No. 10, 31.10.2019, p. 1690-1708.

Research output: Contribution to Journal/MagazineJournal articlepeer-review

Harvard

Mordecai, EA, Caldwell, JM, Grossman, MK, Lippi, CA, Johnson, LR, Neira, M, Rohr, JR, Ryan, SJ, Savage, V, Shocket, MS, Sippy, R, Ibarra, AMS, Thomas, MB, Villena, O & Byers, J (ed.) 2019, 'Thermal biology of mosquito‐borne disease', Ecology Letters, vol. 22, no. 10, pp. 1690-1708. https://doi.org/10.1111/ele.13335

APA

Mordecai, E. A., Caldwell, J. M., Grossman, M. K., Lippi, C. A., Johnson, L. R., Neira, M., Rohr, J. R., Ryan, S. J., Savage, V., Shocket, M. S., Sippy, R., Ibarra, A. M. S., Thomas, M. B., Villena, O., & Byers, J. (Ed.) (2019). Thermal biology of mosquito‐borne disease. Ecology Letters, 22(10), 1690-1708. https://doi.org/10.1111/ele.13335

Vancouver

Mordecai EA, Caldwell JM, Grossman MK, Lippi CA, Johnson LR, Neira M et al. Thermal biology of mosquito‐borne disease. Ecology Letters. 2019 Oct 31;22(10):1690-1708. doi: 10.1111/ele.13335

Author

Mordecai, Erin A. ; Caldwell, Jamie M. ; Grossman, Marissa K. et al. / Thermal biology of mosquito‐borne disease. In: Ecology Letters. 2019 ; Vol. 22, No. 10. pp. 1690-1708.

Bibtex

@article{b49c7eafdeec47409c647fe9e798644e,
title = "Thermal biology of mosquito‐borne disease",
abstract = "Mosquito-borne diseases cause a major burden of disease worldwide. The vital rates of these ectothermic vectors and parasites respond strongly and nonlinearly to temperature and therefore to climate change. Here, we review how trait-based approaches can synthesise and mechanistically predict the temperature dependence of transmission across vectors, pathogens, and environments.We present 11 pathogens transmitted by 15 different mosquito species – including globally important diseases like malaria, dengue, and Zika – synthesised from previously published studies. Transmission varied strongly and unimodally with temperature, peaking at 23–29C and declining to zero below 9–23C and above 32–38C. Different traits restricted transmission at low versus high temperatures, and temperature effects on transmission varied by both mosquito and parasite species. Temperate pathogens exhibit broader thermal ranges and cooler thermal minima and optima than tropical pathogens. Among tropical pathogens, malaria and Ross River virus had lower thermal optima (25–26C) while dengue and Zika viruses had the highest (29C) thermal optima. We expect warming to increase transmission below thermal optima but decrease transmission above optima. Key directions for future work include linking mechanistic models to field transmission, combining temperature effects with control measures, incorporating trait variation and temperature variation, and investigating climate adaptation and migration.",
author = "Mordecai, {Erin A.} and Caldwell, {Jamie M.} and Grossman, {Marissa K.} and Lippi, {Catherine A.} and Johnson, {Leah R.} and Marco Neira and Rohr, {Jason R.} and Ryan, {Sadie J.} and Van Savage and Shocket, {Marta S.} and Rachel Sippy and Ibarra, {Anna M. Stewart} and Thomas, {Matthew B.} and Oswaldo Villena and Byers, {James (Jeb)}",
year = "2019",
month = oct,
day = "31",
doi = "10.1111/ele.13335",
language = "English",
volume = "22",
pages = "1690--1708",
journal = "Ecology Letters",
issn = "1461-023X",
publisher = "Wiley",
number = "10",

}

RIS

TY - JOUR

T1 - Thermal biology of mosquito‐borne disease

AU - Mordecai, Erin A.

AU - Caldwell, Jamie M.

AU - Grossman, Marissa K.

AU - Lippi, Catherine A.

AU - Johnson, Leah R.

AU - Neira, Marco

AU - Rohr, Jason R.

AU - Ryan, Sadie J.

AU - Savage, Van

AU - Shocket, Marta S.

AU - Sippy, Rachel

AU - Ibarra, Anna M. Stewart

AU - Thomas, Matthew B.

AU - Villena, Oswaldo

A2 - Byers, James (Jeb)

PY - 2019/10/31

Y1 - 2019/10/31

N2 - Mosquito-borne diseases cause a major burden of disease worldwide. The vital rates of these ectothermic vectors and parasites respond strongly and nonlinearly to temperature and therefore to climate change. Here, we review how trait-based approaches can synthesise and mechanistically predict the temperature dependence of transmission across vectors, pathogens, and environments.We present 11 pathogens transmitted by 15 different mosquito species – including globally important diseases like malaria, dengue, and Zika – synthesised from previously published studies. Transmission varied strongly and unimodally with temperature, peaking at 23–29C and declining to zero below 9–23C and above 32–38C. Different traits restricted transmission at low versus high temperatures, and temperature effects on transmission varied by both mosquito and parasite species. Temperate pathogens exhibit broader thermal ranges and cooler thermal minima and optima than tropical pathogens. Among tropical pathogens, malaria and Ross River virus had lower thermal optima (25–26C) while dengue and Zika viruses had the highest (29C) thermal optima. We expect warming to increase transmission below thermal optima but decrease transmission above optima. Key directions for future work include linking mechanistic models to field transmission, combining temperature effects with control measures, incorporating trait variation and temperature variation, and investigating climate adaptation and migration.

AB - Mosquito-borne diseases cause a major burden of disease worldwide. The vital rates of these ectothermic vectors and parasites respond strongly and nonlinearly to temperature and therefore to climate change. Here, we review how trait-based approaches can synthesise and mechanistically predict the temperature dependence of transmission across vectors, pathogens, and environments.We present 11 pathogens transmitted by 15 different mosquito species – including globally important diseases like malaria, dengue, and Zika – synthesised from previously published studies. Transmission varied strongly and unimodally with temperature, peaking at 23–29C and declining to zero below 9–23C and above 32–38C. Different traits restricted transmission at low versus high temperatures, and temperature effects on transmission varied by both mosquito and parasite species. Temperate pathogens exhibit broader thermal ranges and cooler thermal minima and optima than tropical pathogens. Among tropical pathogens, malaria and Ross River virus had lower thermal optima (25–26C) while dengue and Zika viruses had the highest (29C) thermal optima. We expect warming to increase transmission below thermal optima but decrease transmission above optima. Key directions for future work include linking mechanistic models to field transmission, combining temperature effects with control measures, incorporating trait variation and temperature variation, and investigating climate adaptation and migration.

U2 - 10.1111/ele.13335

DO - 10.1111/ele.13335

M3 - Journal article

VL - 22

SP - 1690

EP - 1708

JO - Ecology Letters

JF - Ecology Letters

SN - 1461-023X

IS - 10

ER -