Final published version
Research output: Contribution to Journal/Magazine › Journal article › peer-review
| <mark>Journal publication date</mark> | 10/10/2014 |
|---|---|
| <mark>Journal</mark> | Science |
| Issue number | 6206 |
| Volume | 346 |
| Number of pages | 1 |
| Pages (from-to) | 176 |
| Publication Status | Published |
| <mark>Original language</mark> | English |
Tyzio et al. (Reports, 7 February 2014, p. 675) reported that bumetanide restored the impaired oxytocin-mediated γ-aminobutyric acid (GABA) excitatory-inhibitory shift during delivery in animal models of autism, ameliorating some autistic-like characteristics in the offspring. However, standard practices in the study of these models, such as the use of sex-dimorphic or males-only analyses and implementation of tests measuring social behavior, are lacking to definitely associate their findings to autism.