Comment on "Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring"

Biomedical and Life Sciences

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Comment on "Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring". / Bambini-Junior, Victorio; Nunes, Gustavo Della Flora; Schneider, Tomasz et al.
In: Science, Vol. 346, No. 6206, 10.10.2014, p. 176.

Research output: Contribution to Journal/Magazine › Journal article › peer-review

Harvard

Bambini-Junior, V, Nunes, GDF, Schneider, T & Gottfried, C 2014, 'Comment on "Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring"', Science, vol. 346, no. 6206, pp. 176. https://doi.org/10.1126/science.1255679

APA

Bambini-Junior, V., Nunes, G. D. F., Schneider, T., & Gottfried, C. (2014). Comment on "Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring". Science, 346(6206), 176. https://doi.org/10.1126/science.1255679

Vancouver

Bambini-Junior V, Nunes GDF, Schneider T, Gottfried C. Comment on "Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring". Science. 2014 Oct 10;346(6206):176. doi: 10.1126/science.1255679

Author

Bambini-Junior, Victorio ; Nunes, Gustavo Della Flora ; Schneider, Tomasz et al. / Comment on "Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring". In: Science. 2014 ; Vol. 346, No. 6206. pp. 176.

Bibtex

@article{aed5608acec34a3bae7d76e8021b5f08,

title = "Comment on {"}Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring{"}",

abstract = "Tyzio et al. (Reports, 7 February 2014, p. 675) reported that bumetanide restored the impaired oxytocin-mediated γ-aminobutyric acid (GABA) excitatory-inhibitory shift during delivery in animal models of autism, ameliorating some autistic-like characteristics in the offspring. However, standard practices in the study of these models, such as the use of sex-dimorphic or males-only analyses and implementation of tests measuring social behavior, are lacking to definitely associate their findings to autism. ",

keywords = "Animals, Autistic Disorder/chemically induced, Cytoprotection, Female, Oxytocin/metabolism, Pregnancy, gamma-Aminobutyric Acid/metabolism",

author = "Victorio Bambini-Junior and Nunes, {Gustavo Della Flora} and Tomasz Schneider and Carmem Gottfried",

note = "Copyright {\textcopyright} 2014, American Association for the Advancement of Science.",

year = "2014",

month = oct,

day = "10",

doi = "10.1126/science.1255679",

language = "English",

volume = "346",

pages = "176",

journal = "Science",

issn = "0036-8075",

publisher = "American Association for the Advancement of Science",

number = "6206",

}

RIS

TY - JOUR

T1 - Comment on "Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring"

AU - Bambini-Junior, Victorio

AU - Nunes, Gustavo Della Flora

AU - Schneider, Tomasz

AU - Gottfried, Carmem

PY - 2014/10/10

Y1 - 2014/10/10

N2 - Tyzio et al. (Reports, 7 February 2014, p. 675) reported that bumetanide restored the impaired oxytocin-mediated γ-aminobutyric acid (GABA) excitatory-inhibitory shift during delivery in animal models of autism, ameliorating some autistic-like characteristics in the offspring. However, standard practices in the study of these models, such as the use of sex-dimorphic or males-only analyses and implementation of tests measuring social behavior, are lacking to definitely associate their findings to autism.

AB - Tyzio et al. (Reports, 7 February 2014, p. 675) reported that bumetanide restored the impaired oxytocin-mediated γ-aminobutyric acid (GABA) excitatory-inhibitory shift during delivery in animal models of autism, ameliorating some autistic-like characteristics in the offspring. However, standard practices in the study of these models, such as the use of sex-dimorphic or males-only analyses and implementation of tests measuring social behavior, are lacking to definitely associate their findings to autism.

KW - Animals

KW - Autistic Disorder/chemically induced

KW - Cytoprotection

KW - Female

KW - Oxytocin/metabolism

KW - Pregnancy

KW - gamma-Aminobutyric Acid/metabolism

U2 - 10.1126/science.1255679

DO - 10.1126/science.1255679

M3 - Journal article

C2 - 25301610

VL - 346

SP - 176

JO - Science

JF - Science

SN - 0036-8075

IS - 6206

ER -

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