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Elk-1 associates with FAK, regulates the expression of FAK and MAP kinases as well as apoptosis in HK-2 cells

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Elk-1 associates with FAK, regulates the expression of FAK and MAP kinases as well as apoptosis in HK-2 cells. / Mamali, I.; Kotsantis, P.; Lampropoulou, M. et al.
In: Journal of Cellular Physiology, Vol. 216, No. 1, 31.07.2008, p. 198-206.

Research output: Contribution to Journal/MagazineJournal articlepeer-review

Harvard

Mamali, I, Kotsantis, P, Lampropoulou, M & Marmaras, VJ 2008, 'Elk-1 associates with FAK, regulates the expression of FAK and MAP kinases as well as apoptosis in HK-2 cells', Journal of Cellular Physiology, vol. 216, no. 1, pp. 198-206. https://doi.org/10.1002/jcp.21390

APA

Mamali, I., Kotsantis, P., Lampropoulou, M., & Marmaras, V. J. (2008). Elk-1 associates with FAK, regulates the expression of FAK and MAP kinases as well as apoptosis in HK-2 cells. Journal of Cellular Physiology, 216(1), 198-206. https://doi.org/10.1002/jcp.21390

Vancouver

Mamali I, Kotsantis P, Lampropoulou M, Marmaras VJ. Elk-1 associates with FAK, regulates the expression of FAK and MAP kinases as well as apoptosis in HK-2 cells. Journal of Cellular Physiology. 2008 Jul 31;216(1):198-206. Epub 2008 Feb 4. doi: 10.1002/jcp.21390

Author

Mamali, I. ; Kotsantis, P. ; Lampropoulou, M. et al. / Elk-1 associates with FAK, regulates the expression of FAK and MAP kinases as well as apoptosis in HK-2 cells. In: Journal of Cellular Physiology. 2008 ; Vol. 216, No. 1. pp. 198-206.

Bibtex

@article{5b1c556602824e0bad860943a15963ff,
title = "Elk-1 associates with FAK, regulates the expression of FAK and MAP kinases as well as apoptosis in HK-2 cells",
abstract = "Focal adhesion kinase (FAK), MAP kinases and the nuclear transcription factor Elk-1 have been reported to be implicated in the same cellular processes, however, their direct or indirect interaction and potential function(s) has not been documented. Here, we explored the association of FAK with Elk-1, the implication of Elk-1 in the regulation of FAK and MAP kinases expression as well as apoptosis, in HK-2 cells. Biochemical and immunofluorescence approaches strongly support the association of low molecular weight protein bands, recognized by FAK antibodies, with Elk-1 or pser383Elk-1. The FAK/Elk-1 complex is found, mainly, in the cytoplasm, near the nuclear membrane periphery, raising the possibility that Elk-1 may have alternative extranuclear function(s) in HK-2 cells. Furthermore, we demonstrated that Elk-1 siRNA-mediated knockdown experiments, increased apoptosis. By contrast, Elk-1 siRNA decreased significantly the expression of FAK and MAP kinases, supporting the hypothesis that Elk-1 may act as a potential physiological substrate and regulator of FAK and MAP kinases expression. These results strongly support that Elk-1 protein is a novel binding-protein partner for FAK, a finding that significantly broadens the potential functioning of FAK and Elk-1. J. Cell. Physiol. 216: 198–206, 2008.",
author = "I. Mamali and P. Kotsantis and M. Lampropoulou and V.J. Marmaras",
year = "2008",
month = jul,
day = "31",
doi = "10.1002/jcp.21390",
language = "English",
volume = "216",
pages = "198--206",
journal = "Journal of Cellular Physiology",
number = "1",

}

RIS

TY - JOUR

T1 - Elk-1 associates with FAK, regulates the expression of FAK and MAP kinases as well as apoptosis in HK-2 cells

AU - Mamali, I.

AU - Kotsantis, P.

AU - Lampropoulou, M.

AU - Marmaras, V.J.

PY - 2008/7/31

Y1 - 2008/7/31

N2 - Focal adhesion kinase (FAK), MAP kinases and the nuclear transcription factor Elk-1 have been reported to be implicated in the same cellular processes, however, their direct or indirect interaction and potential function(s) has not been documented. Here, we explored the association of FAK with Elk-1, the implication of Elk-1 in the regulation of FAK and MAP kinases expression as well as apoptosis, in HK-2 cells. Biochemical and immunofluorescence approaches strongly support the association of low molecular weight protein bands, recognized by FAK antibodies, with Elk-1 or pser383Elk-1. The FAK/Elk-1 complex is found, mainly, in the cytoplasm, near the nuclear membrane periphery, raising the possibility that Elk-1 may have alternative extranuclear function(s) in HK-2 cells. Furthermore, we demonstrated that Elk-1 siRNA-mediated knockdown experiments, increased apoptosis. By contrast, Elk-1 siRNA decreased significantly the expression of FAK and MAP kinases, supporting the hypothesis that Elk-1 may act as a potential physiological substrate and regulator of FAK and MAP kinases expression. These results strongly support that Elk-1 protein is a novel binding-protein partner for FAK, a finding that significantly broadens the potential functioning of FAK and Elk-1. J. Cell. Physiol. 216: 198–206, 2008.

AB - Focal adhesion kinase (FAK), MAP kinases and the nuclear transcription factor Elk-1 have been reported to be implicated in the same cellular processes, however, their direct or indirect interaction and potential function(s) has not been documented. Here, we explored the association of FAK with Elk-1, the implication of Elk-1 in the regulation of FAK and MAP kinases expression as well as apoptosis, in HK-2 cells. Biochemical and immunofluorescence approaches strongly support the association of low molecular weight protein bands, recognized by FAK antibodies, with Elk-1 or pser383Elk-1. The FAK/Elk-1 complex is found, mainly, in the cytoplasm, near the nuclear membrane periphery, raising the possibility that Elk-1 may have alternative extranuclear function(s) in HK-2 cells. Furthermore, we demonstrated that Elk-1 siRNA-mediated knockdown experiments, increased apoptosis. By contrast, Elk-1 siRNA decreased significantly the expression of FAK and MAP kinases, supporting the hypothesis that Elk-1 may act as a potential physiological substrate and regulator of FAK and MAP kinases expression. These results strongly support that Elk-1 protein is a novel binding-protein partner for FAK, a finding that significantly broadens the potential functioning of FAK and Elk-1. J. Cell. Physiol. 216: 198–206, 2008.

U2 - 10.1002/jcp.21390

DO - 10.1002/jcp.21390

M3 - Journal article

VL - 216

SP - 198

EP - 206

JO - Journal of Cellular Physiology

JF - Journal of Cellular Physiology

IS - 1

ER -