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Low-frequency oscillations of the laser Doppler perfusion signal in human skin

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Low-frequency oscillations of the laser Doppler perfusion signal in human skin. / Kvandal, Per; Landsverk, Svein Aslak; Bernjak, Alan et al.
In: Microvascular Research, Vol. 72, No. 3, 01.11.2006, p. 120-127.

Research output: Contribution to Journal/MagazineJournal articlepeer-review

Harvard

Kvandal, P, Landsverk, SA, Bernjak, A, Stefanovska, A, Kvernmo, HD & Kirkebøen, KA 2006, 'Low-frequency oscillations of the laser Doppler perfusion signal in human skin', Microvascular Research, vol. 72, no. 3, pp. 120-127. https://doi.org/10.1016/j.mvr.2006.05.006

APA

Kvandal, P., Landsverk, S. A., Bernjak, A., Stefanovska, A., Kvernmo, H. D., & Kirkebøen, K. A. (2006). Low-frequency oscillations of the laser Doppler perfusion signal in human skin. Microvascular Research, 72(3), 120-127. https://doi.org/10.1016/j.mvr.2006.05.006

Vancouver

Kvandal P, Landsverk SA, Bernjak A, Stefanovska A, Kvernmo HD, Kirkebøen KA. Low-frequency oscillations of the laser Doppler perfusion signal in human skin. Microvascular Research. 2006 Nov 1;72(3):120-127. doi: 10.1016/j.mvr.2006.05.006

Author

Kvandal, Per ; Landsverk, Svein Aslak ; Bernjak, Alan et al. / Low-frequency oscillations of the laser Doppler perfusion signal in human skin. In: Microvascular Research. 2006 ; Vol. 72, No. 3. pp. 120-127.

Bibtex

@article{71b1f32045574f8bb89b398cfea9133a,
title = "Low-frequency oscillations of the laser Doppler perfusion signal in human skin",
abstract = "Spectral analysis of the laser Doppler flow (LDF) signal in the frequency interval from 0.0095-2.0 Hz reveals blood flow oscillations with frequencies around 1.0, 0.3, 0.1, 0.04 and 0.01 Hz. The heartbeat, the respiration, the intrinsic myogenic activity of vascular smooth muscle, the neurogenic activity of the vessel wall and the vascular endothelium influence these oscillations, respectively. The first aim of this study was to investigate if a slow oscillatory component could be detected in the frequency area below 0.0095 Hz of the human cutaneous blood perfusion signal. Unstimulated basal blood skin perfusion and enhanced perfusion during iontophoresis with the endothelium-dependent vasodilator acetylcholine (ACh) and the endothelium-independent vasodilator sodium nitroprusside (SNP) were measured in healthy male volunteers and the wavelet transform was computed. A low-frequency oscillation between 0.005 and 0.0095 Hz was found both during basal conditions and during iontophoresis with ACh and SNP. Iontophoresis with ACh increased the normalized amplitude to a greater extent than SNP (P  =  0.001) indicating modulation by the vascular endothelium. To gain further insight into the mechanisms for this endothelium dependency, we inhibited nitric oxide (NO) synthesis with NG-monomethyl-l-arginine (l-NMMA) and prostaglandin (PG) synthesis by aspirin. l-NMMA did not affect the increased response to ACh vs. SNP iontophoresis in the 0.005-0.0095-Hz interval (P  =  0.006) but abolished the difference in the 0.0095-0.021-Hz interval (P  =  0.97). Aspirin did not affect the difference in response to ACh and SNP in either of the two frequency intervals. Thus, other endothelial mechanisms, such as endothelium-derived hyperpolarizing factor (EDHF), might be involved in the regulation of this sixth frequency interval (0.005-0.0095 Hz).",
keywords = "Acetylcholine, endothelium-derived hyperpolarizing factor, Endothelium-mediated vasodilatation, Nitric oxide, Oscillations, Spectral analysis, Wavelet transform",
author = "Per Kvandal and Landsverk, {Svein Aslak} and Alan Bernjak and Aneta Stefanovska and Kvernmo, {Hebe D{\'e}sir{\'e}e} and Kirkeb{\o}en, {Knut Arvid}",
year = "2006",
month = nov,
day = "1",
doi = "10.1016/j.mvr.2006.05.006",
language = "English",
volume = "72",
pages = "120--127",
journal = "Microvascular Research",
issn = "0026-2862",
publisher = "Academic Press Inc.",
number = "3",

}

RIS

TY - JOUR

T1 - Low-frequency oscillations of the laser Doppler perfusion signal in human skin

AU - Kvandal, Per

AU - Landsverk, Svein Aslak

AU - Bernjak, Alan

AU - Stefanovska, Aneta

AU - Kvernmo, Hebe Désirée

AU - Kirkebøen, Knut Arvid

PY - 2006/11/1

Y1 - 2006/11/1

N2 - Spectral analysis of the laser Doppler flow (LDF) signal in the frequency interval from 0.0095-2.0 Hz reveals blood flow oscillations with frequencies around 1.0, 0.3, 0.1, 0.04 and 0.01 Hz. The heartbeat, the respiration, the intrinsic myogenic activity of vascular smooth muscle, the neurogenic activity of the vessel wall and the vascular endothelium influence these oscillations, respectively. The first aim of this study was to investigate if a slow oscillatory component could be detected in the frequency area below 0.0095 Hz of the human cutaneous blood perfusion signal. Unstimulated basal blood skin perfusion and enhanced perfusion during iontophoresis with the endothelium-dependent vasodilator acetylcholine (ACh) and the endothelium-independent vasodilator sodium nitroprusside (SNP) were measured in healthy male volunteers and the wavelet transform was computed. A low-frequency oscillation between 0.005 and 0.0095 Hz was found both during basal conditions and during iontophoresis with ACh and SNP. Iontophoresis with ACh increased the normalized amplitude to a greater extent than SNP (P  =  0.001) indicating modulation by the vascular endothelium. To gain further insight into the mechanisms for this endothelium dependency, we inhibited nitric oxide (NO) synthesis with NG-monomethyl-l-arginine (l-NMMA) and prostaglandin (PG) synthesis by aspirin. l-NMMA did not affect the increased response to ACh vs. SNP iontophoresis in the 0.005-0.0095-Hz interval (P  =  0.006) but abolished the difference in the 0.0095-0.021-Hz interval (P  =  0.97). Aspirin did not affect the difference in response to ACh and SNP in either of the two frequency intervals. Thus, other endothelial mechanisms, such as endothelium-derived hyperpolarizing factor (EDHF), might be involved in the regulation of this sixth frequency interval (0.005-0.0095 Hz).

AB - Spectral analysis of the laser Doppler flow (LDF) signal in the frequency interval from 0.0095-2.0 Hz reveals blood flow oscillations with frequencies around 1.0, 0.3, 0.1, 0.04 and 0.01 Hz. The heartbeat, the respiration, the intrinsic myogenic activity of vascular smooth muscle, the neurogenic activity of the vessel wall and the vascular endothelium influence these oscillations, respectively. The first aim of this study was to investigate if a slow oscillatory component could be detected in the frequency area below 0.0095 Hz of the human cutaneous blood perfusion signal. Unstimulated basal blood skin perfusion and enhanced perfusion during iontophoresis with the endothelium-dependent vasodilator acetylcholine (ACh) and the endothelium-independent vasodilator sodium nitroprusside (SNP) were measured in healthy male volunteers and the wavelet transform was computed. A low-frequency oscillation between 0.005 and 0.0095 Hz was found both during basal conditions and during iontophoresis with ACh and SNP. Iontophoresis with ACh increased the normalized amplitude to a greater extent than SNP (P  =  0.001) indicating modulation by the vascular endothelium. To gain further insight into the mechanisms for this endothelium dependency, we inhibited nitric oxide (NO) synthesis with NG-monomethyl-l-arginine (l-NMMA) and prostaglandin (PG) synthesis by aspirin. l-NMMA did not affect the increased response to ACh vs. SNP iontophoresis in the 0.005-0.0095-Hz interval (P  =  0.006) but abolished the difference in the 0.0095-0.021-Hz interval (P  =  0.97). Aspirin did not affect the difference in response to ACh and SNP in either of the two frequency intervals. Thus, other endothelial mechanisms, such as endothelium-derived hyperpolarizing factor (EDHF), might be involved in the regulation of this sixth frequency interval (0.005-0.0095 Hz).

KW - Acetylcholine

KW - endothelium-derived hyperpolarizing factor

KW - Endothelium-mediated vasodilatation

KW - Nitric oxide

KW - Oscillations

KW - Spectral analysis

KW - Wavelet transform

U2 - 10.1016/j.mvr.2006.05.006

DO - 10.1016/j.mvr.2006.05.006

M3 - Journal article

C2 - 16854436

AN - SCOPUS:33750619115

VL - 72

SP - 120

EP - 127

JO - Microvascular Research

JF - Microvascular Research

SN - 0026-2862

IS - 3

ER -