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Osteopontin as a Biomarker for Coronary Artery Disease

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Osteopontin as a Biomarker for Coronary Artery Disease. / Layton, G.R.; Antoun, I.; Copperwheat, A. et al.
In: Cells, Vol. 14, No. 2, 13.01.2025.

Research output: Contribution to Journal/MagazineJournal articlepeer-review

Harvard

Layton, GR, Antoun, I, Copperwheat, A, Khan, ZL, Bhandari, SS, Somani, R, Ng, A & Zakkar, M 2025, 'Osteopontin as a Biomarker for Coronary Artery Disease', Cells, vol. 14, no. 2. https://doi.org/10.3390/cells14020106

APA

Layton, G. R., Antoun, I., Copperwheat, A., Khan, Z. L., Bhandari, S. S., Somani, R., Ng, A., & Zakkar, M. (2025). Osteopontin as a Biomarker for Coronary Artery Disease. Cells, 14(2). https://doi.org/10.3390/cells14020106

Vancouver

Layton GR, Antoun I, Copperwheat A, Khan ZL, Bhandari SS, Somani R et al. Osteopontin as a Biomarker for Coronary Artery Disease. Cells. 2025 Jan 13;14(2). doi: 10.3390/cells14020106

Author

Layton, G.R. ; Antoun, I. ; Copperwheat, A. et al. / Osteopontin as a Biomarker for Coronary Artery Disease. In: Cells. 2025 ; Vol. 14, No. 2.

Bibtex

@article{10c1f42dbcfe4e5891ad2c821f1bb460,
title = "Osteopontin as a Biomarker for Coronary Artery Disease",
abstract = "Osteopontin (OPN) is a sialylated phosphoprotein highly expressed in atherosclerosis and upregulated in settings of both acute and chronic inflammation. It is hypothesised that plasma levels of OPN may correlate with the presence of coronary artery disease, “CAD”. This offers potential as a point-of-care testing biomarker for early diagnosis, disease monitoring, and prognosis. This review evaluates the current literature on the association between plasma OPN levels and coronary artery disease and what is currently known to support its potential as a biomarker for future practice. Electronic searches of MEDLINE and EMBASE databases were undertaken from inception until July 2024. Thirty-three studies met the inclusion criteria. All studies were observational, with gross heterogeneity in methods used to analyse the association of plasma OPN with clinical characteristics. They included case series, case–control, cross-sectional, and cohort study designs. OPN has been linked to higher cardiovascular risk and unfavourable cardiovascular outcomes. However, the evidence regarding the direct assessment of CAD severity using tools like the SYNTAX or TIMI scores, which focus on anatomical complexity and risk factors, is less definitive. This suggests that OPN may be a more precise reflection of the inflammatory processes and atherosclerotic activity contributing to unfavourable outcomes rather than a direct indicator of the anatomical severity of CAD itself. Consequently, OPN is increasingly perceived as a marker of a poor prognosis rather than a tool for assessing the severity of coronary artery lesions.",
author = "G.R. Layton and I. Antoun and A. Copperwheat and Z.L. Khan and S.S. Bhandari and R. Somani and A. Ng and M. Zakkar",
year = "2025",
month = jan,
day = "13",
doi = "10.3390/cells14020106",
language = "English",
volume = "14",
journal = "Cells",
issn = "2073-4409",
publisher = "Multidisciplinary Digital Publishing Institute (MDPI)",
number = "2",

}

RIS

TY - JOUR

T1 - Osteopontin as a Biomarker for Coronary Artery Disease

AU - Layton, G.R.

AU - Antoun, I.

AU - Copperwheat, A.

AU - Khan, Z.L.

AU - Bhandari, S.S.

AU - Somani, R.

AU - Ng, A.

AU - Zakkar, M.

PY - 2025/1/13

Y1 - 2025/1/13

N2 - Osteopontin (OPN) is a sialylated phosphoprotein highly expressed in atherosclerosis and upregulated in settings of both acute and chronic inflammation. It is hypothesised that plasma levels of OPN may correlate with the presence of coronary artery disease, “CAD”. This offers potential as a point-of-care testing biomarker for early diagnosis, disease monitoring, and prognosis. This review evaluates the current literature on the association between plasma OPN levels and coronary artery disease and what is currently known to support its potential as a biomarker for future practice. Electronic searches of MEDLINE and EMBASE databases were undertaken from inception until July 2024. Thirty-three studies met the inclusion criteria. All studies were observational, with gross heterogeneity in methods used to analyse the association of plasma OPN with clinical characteristics. They included case series, case–control, cross-sectional, and cohort study designs. OPN has been linked to higher cardiovascular risk and unfavourable cardiovascular outcomes. However, the evidence regarding the direct assessment of CAD severity using tools like the SYNTAX or TIMI scores, which focus on anatomical complexity and risk factors, is less definitive. This suggests that OPN may be a more precise reflection of the inflammatory processes and atherosclerotic activity contributing to unfavourable outcomes rather than a direct indicator of the anatomical severity of CAD itself. Consequently, OPN is increasingly perceived as a marker of a poor prognosis rather than a tool for assessing the severity of coronary artery lesions.

AB - Osteopontin (OPN) is a sialylated phosphoprotein highly expressed in atherosclerosis and upregulated in settings of both acute and chronic inflammation. It is hypothesised that plasma levels of OPN may correlate with the presence of coronary artery disease, “CAD”. This offers potential as a point-of-care testing biomarker for early diagnosis, disease monitoring, and prognosis. This review evaluates the current literature on the association between plasma OPN levels and coronary artery disease and what is currently known to support its potential as a biomarker for future practice. Electronic searches of MEDLINE and EMBASE databases were undertaken from inception until July 2024. Thirty-three studies met the inclusion criteria. All studies were observational, with gross heterogeneity in methods used to analyse the association of plasma OPN with clinical characteristics. They included case series, case–control, cross-sectional, and cohort study designs. OPN has been linked to higher cardiovascular risk and unfavourable cardiovascular outcomes. However, the evidence regarding the direct assessment of CAD severity using tools like the SYNTAX or TIMI scores, which focus on anatomical complexity and risk factors, is less definitive. This suggests that OPN may be a more precise reflection of the inflammatory processes and atherosclerotic activity contributing to unfavourable outcomes rather than a direct indicator of the anatomical severity of CAD itself. Consequently, OPN is increasingly perceived as a marker of a poor prognosis rather than a tool for assessing the severity of coronary artery lesions.

U2 - 10.3390/cells14020106

DO - 10.3390/cells14020106

M3 - Journal article

VL - 14

JO - Cells

JF - Cells

SN - 2073-4409

IS - 2

ER -